Insomnia Self Care

Common Types

We offer the following Product introduce:

Ces Device for Anxiety
Through indirect stimulation of brain tissue in the hypothalamic region, CES (Cranial Electrotherapy Stimulation) devices utilize low-frequency electrical pulses to induce relaxation by transmitting signals to the brain.

Non Pharmacological Treatment for Insomnia
Sleeplessness starts in your brain, where electrochemical signals called neurotransmitters control whether we are asleep or awake. CES safely modulates the brain's electrochemical signals, bringing these signals back into balance with electrotherapy.

Ces Therapy for Depression
The therapy delivers microcurrent pulses (typically 0.5-100Hz) through electrodes placed on the head
These signals stimulate the hypothalamus and limbic system. This stimulation helps normalize production of key neurotransmitters:
• Serotonin (mood regulation)
• Dopamine (pleasure/reward)
• GABA (calming effect)
• Beta-endorphins (natural pain relief)

Ces Therapy Device
Cranial Electrotherapy Stimulation (CES) is a therapeutic intervention involving gentle electrical stimulation directed at the brain to alleviate symptoms of depression and anxiety. By indirectly activating brain tissue in the hypothalamic region, CES aims to prompt the restoration of neurochemical equilibrium, returning the brain to a pre-stress state. Clinical research attests to its safety and efficacy, with many individuals observing positive outcomes within a few days of commencing treatment.

CES therapy device Clinical Study

Regarding the pathway of current transmission to the brain, the utilization of earlobe electrodes in this investigation suggests that the alternating microcurrent initially activates afferent branches of cranial nerves. Stimulation likely commences at facial, glossopharyngeal, or vagus nerve branches near the electrode placement on the earlobe, subsequently traveling to the brainstem, thalamus, and eventually reaching the cortex.

Two distinct frequencies, 100 or 0.5 Hz, demonstrated efficacy in brain deactivation, irrespective of current amplitude. This offers supplementary mechanistic insight suggesting that CES may modulate cortical activity by disrupting typical brain oscillation patterns, potentially through the introduction of high- or low-frequency noise.

The findings of this study carry significant clinical implications. Application of alternating current (AC) at varying frequencies could potentially modify communication between nodes of the default mode network (DMN). Research in clinical cohorts, encompassing anxiety disorders and depression, has revealed aberrations within these intrinsic connectivity networks. For instance, individuals with anxiety disorders exhibit diminished activity in DMN-associated regions like the posterior cingulate cortex (PCC), inferior parietal lobule, medial prefrontal cortex, and thalamus when confronted with threat-related stimuli. Moreover, reduced functional connectivity within the sensorimotor network (SMN) and DMN has been documented in individuals with social anxiety disorder by Liao et al. Furthermore, both acute and chronic pain sufferers exhibit abnormal functional connectivity within the DMN.